By Andrew Hadley, Peter Soothill
The potent prevention, prognosis, and administration of alloimmune cytopenias has turn into a staff attempt concerning hematologists, obstetricians, pediatricians, immunologists, laboratory technicians, midwives, and study scientists. This booklet has been written by way of specialists of their respective fields to assemble the problems of pathogenesis, epidemiology, prevention, prognosis, and medical administration. This accomplished yet obtainable account is greatly cross-referenced to stress the hyperlinks among pathogenesis and scientific sequels, among epidemiology and the reason for screening courses, and among prognosis and healing intervention.
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Additional info for Alloimmune Disorders of Pregnancy: Anaemia, Thrombocytopenia and Neutropenia in the Fetus and Newborn
Nevertheless, future diagnostic and therapeutic advances depend on an improved understanding of the molecular basis of the maternal alloimmune response and the processes involved in the immune destruction of blood cells in the fetus. Progress in these areas should lead to the development of new strategies to modulate maternal immune responses and speciﬁcally suppress the destruction of fetal cells. Transgenic animal technology may provide one important means of establishing improved models with which to study these processes and evaluate the potential of new therapies.
At its most extreme, it culminates in fetal or neonatal death, yet in many publications antibodies have been reported to have caused HDFN when the indications have been no more than a positive direct antiglobulin test result with fetal red cells. In this chapter, HDFN will generally only be considered to have occurred when clinical intervention in excess of phototherapy for jaundice, such as intrauterine or neonatal transfusion, was indicated, or when kernicterus or hydrops was reported. Multifarious red cell antigens will be mentioned in this chapter and it is not possible to refer to the original publications describing all of them.
Transfusion Medicine, 4, 25–33. 69 Murphy FJ & Reen DJ (1996). Diﬀerential expression of function-related antigens on newborn and adult monocyte subpopulations. Immunology, 89, 587–91. 70 Warmerdam PAM, van der Winkel JGJ, Gosselin EJ & Capel PJA (1990). Molecular basis for a polymorphism of human Fc␥RII (CD32). Journal of Experimental Medicine, 172, 19–25. 71 Denomme G, Ryan G & Fernandes B (1997). The Fc␥RIIa-His131 allotype is overexpressed in infants with ABO hemolytic disease of the newborn.