By F. Lang, M. Ritter, H. Völkl, D. Häussinger (auth.), Prof. Dr. Florian Lang, Prof. Dr. Dieter Häussinger (eds.)
One of the necessities for survival is the power of cells to take care of their quantity. therefore, through the process evolution cells have "learned" various options to accomplish quantity homeostasis. This quantity regulatory equipment includes rules of either, mobile metabolism and mobile shipping and is exploited through hormones and transmitters to manage cellularfunction. This publication to illustrates the advanced interaction of mobile quantity regulatory mechanisms and mobile functionality in various tissues. notwithstanding, our wisdom continues to be faraway from being conclusive, and the current number of experiences is assumed to foster extra experimental efforts to resolve the function of cellphone quantity within the built-in functionality of cells.
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Additional info for Advances in Comparative and Environmental Physiology: Interaction of Cell Volume and Cell Function
Adenosine leads to cell shrinkage, whereas phenylephrine slightly increases liver cell volume. The effects of a variety of hormones on liver cell volume are summarized in Table 1. Cell volume modulation by these hormones at least in part involves alterations of cellular K+ and Na+ balance and the cell volume effects exerted by different hormones are largely additive (vom Dahl et al. 1991c). It should be emphasized that cell volume changes markedly under the influence of hormones. For example, insulin plus phenylephrine induces a 20% cell swelling, whereas vasopressin plus glucagon leads to about 20% cell shrinkage.
1989; Haussinger et al. 1990d). The release of potassium is inhibited by barium, quinidine and SITS (Haddad et al. 1989; Haussinger et al. 1990d). Accordingly, cell swelling apparently leads to activation of potassium channels in parallel to anion channels. In isolated hepatocytes, RVD has similarly been inhibited by barium and quinidine (Corasanti et al. 1990b). The activation of the K+ channels leads to the respective hyperpolarization of the cell membrane (Graf et al. 1988). Hepatocyte swelling apparently opens stretch-activated nonselective cation channels, which allow passage of Ca2 + into the cell (Bear 1990).
Lang et al. Ubi J, Murer H, Kolb H-A (1988b) Ion channels activated by osmotic and mechanical stress in membranes of opossum kidney cells. J Membr Bioi 104: 223-232 Ullrich KJ, Jarausch KH (1956) Untersuchungen zum Problem der Harnkonzentrierung und Harnverdiinnung. Pfliigers Arch Eur J Physiol 262: 537-550 Ussing HH (1965) Relationship between osmotic reactions and active sodium transport in frog skin epithelium. Acta Physiol Scand 63: 141-155 Ussing HH (1985) Volume regulation and basolateral cotransport of sodium, potassium, and chloride ions in frog skin epithelium.